Post traumatic neuropathic facial pain

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These forms of TN should be distinguished from idiopathic atypical facial painas well as other disorders causing cranio-facial pain. Nearly all cases of typical TN are caused by blood vessels compressing the trigeminal nerve root as it enters the brain stem. This neurovascular or microvascular compression at the trigeminal nerve root entry zone may be caused by arteries of veins, large or small, that may simply contact or indent the trigeminal nerve.

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The most widely accepted view is that myelin damage results from irritation of the nerve, usually a blood vessel that causes the nerve to be compressed. Facial pain resulting from unintentional injury to the trigeminal system from facial trauma, oral surgery, ear, nose and throat ENT surgery, root injury from posterior fossa or skull base surgery, stroke, etc. This pain is described as dull, burning, or boring and is usually constant because the injured nerve spontaneously sends impulses to the brain.

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Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct. Readers must therefore always check the product information and clinical procedures with the most up to date published product information and data sheets provided by the manufacturers and the most recent codes of conduct and safety regulations. The authors and the publishers do not accept responsibility or legal liability for any errors in the text or for the misuse or misapplication of material in this work.

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Pregabalin is effective in the treatment of peripheral and central neuropathic pain. This study evaluated the effectiveness of pregablin in management of post traumatic peripheral nerve injury facial pain not responding to other medication like analgesics. Pregabalin was well tolerated. The most common adverse effects were dizziness and tiredness.

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Master in Oral Surgery and Implantology. Valencia University Medical and Dental School. Valencia Spain.

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PMID: An unusual event recently occurred in the field of chronic orofacial pain. A new entity has been established through a few research papers and meetings of experts. Different specialists have known for some time that surgery and other traumatic events may injure the trigeminal nerve and provoke symptoms.

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Peripheral nerve injuries caused by accidental trauma, surgeries or diseases, may evolve to persistent, severe and refractory neuropathic pain, being a major economic and social problem because it often affects most productive population group causing sometimes devastating incapacities. In this brief review, aspects of the prevalence of neuropathic pain by trauma injury of peripheral nerves and its treatment will be evaluated. After evaluating neuropathic pain pathophysiology after peripheral nerve injury, the incidence of peripheral nerve trauma injury and of postoperative chronic pain, of predictive factors and of postoperative neuropathic pain prevention, pharmacological and non-pharmacological treatment of post-trauma and postoperative painful neuropathy are appreciated.

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The traumatic event may be mechanical, chemical, thermal or caused by radiation. Neuroablative procedures for trigeminal neuralgia, aimed at the trigeminal ganglion or nerve root, may result in neuropathic pain involving one or more trigeminal divisions; this should be considered as post-traumatic and coded here. Pain duration ranges widely from paroxysmal to constant, and may be mixed.

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When refering to evidence in academic writing, you should always try to reference the primary original source. That is usually the journal article where the information was first stated. In most cases Physiopedia articles are a secondary source and so should not be used as references.

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In two ambispective clinical trials, we demonstrated that phenotypic differences exist between individuals with neuropathic pain and those without neuropathic pain of the trigeminal nerve. The recurrence of neuropathic pain after trigeminal microsurgery is likely multifactorial and might not depend on factors that normally affect useful or functional sensory recovery in those who have no neuropathic pain. These results indicate that the understanding of post-traumatic trigeminal neuropathic pain is incomplete. Until then, non-surgical treatment for post-traumatic trigeminal neuropathic pain remains a safer option.

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